This pocket-sized Thieme flexibook bargains speedy, trustworthy rationalization of a large and infrequently complicated array of offering signs. The booklet offers important diagnostic details in a handy tabular layout that leaves "no stone unturned" in contemplating the rarer percentages, and is drastically invaluable in attaining a correct analysis. convenient and accomplished, it truly is perfect for physicians interested in reading and admitting sufferers who require neurosurgical intervention.

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Extra info for Differential Diagnosis in Neurology and Neurosurgery Author Sotirios A Tsementzis

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Generally, there is no treatment for atrophy, whereas hydrocephalus can often be treated with ventricular or subarachnoid space shunts and/or removal of the obstructive or overproducing lesion. The diagnosis of NPH requires very close correlation between the clinical findings and the imaging results, and the best diagnostic test for NPH is still clinical improvement after ventricular shunting. It is difficult to distinguish NPH from atrophic ventriculomegaly on a single examination. Follow-up with serial CT or MR imaging is therefore necessary, and may show that the dilated ventricles have returned to normal size, remain enlarged, or, most importantly, that there has been no further interval enlargement.

Usage subject to terms and conditions of license. 42 Neuroradiology T1WI T2WI acute hemorrhage (deoxyhemoglobin) early methemoglobin formation a basilar artery edema +serum subarachnoid space cerebellum intracellular deoxyhemoglobin intracellular methemoglobin stage b early subacute hemorrhage (deoxyhemoglobin) deoxyhemoglobin edema + serum from retracted clot intracellular methemoglobin stage (hemorrhage in acute stage) methemoglobin residual deoxyhemoglobin edema + serum (from retracted clot) hemosiderin and ferritin c edema + serum (from retracted clot) free extracellular methemoglobin edema + serum residual deoxyhemoglobin free methemoglobin d free extracellular methemoglobin hemosiderin and ferritin Fig.

40 Neuroradiology Fig. 3 Posterior fossa lesions 1. Medulloblastoma. Axial MRI T1 WI shows a solid space-occuping lesion with a moderate signal intensity on T2 WI which occupies the area behind the 4th ventricle exerting pressure on it. 2. Ependymoma. Axial MRI T1 WI shows a multilobular space-occuping lesion with solid features, which are enhanced without homogeneity, and cystic features in the periphery and focal calcifications. 3. Pilocytic astrocytoma of the brain stem on axial MRI T1 WI with well-delineated margins and a highly pathological signal; mild compression on the 4th ventricle.

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