By Alberto J. Espay MD MSc, José Biller MD FACP FAAN FAHA

Concise Neurology provides neurology content material in a logical series inside of each one of ten chapters. nearly each subject of curiosity to clinicians is roofed in a single or pages to facilitate effective evaluation of the center options in analysis and therapy for every syndrome or affliction. the writer has maintained a thematic solidarity throughout comparable pages, which permits for random studying to be virtually as fruitful as sequential interpreting. for instance, a reader who opens the "anticoagulation" web page will discover a capsulized evaluation of the heparin, warfarin, and thrombolysis stroke trials and the proof aiding the present healing instructions. Flipping via neighboring pages will swiftly refresh the placement of the clinically correct thrombophilias when it comes to the coagulation pathway, symptoms for lifelong anticoagulation, motives of hyperhomocysteinemia, and the naked bones of an important etiologies of strokes within the young.
 
The ebook provides greater point content material than the scholar "In a web page" textual content, covers every one subject in additional intensity, and addresses concerns concerning pathophysiology and neuropathology of illnesses. Concise Neurology might be a welcome source for busy practitioners, citizens, fellows, scientific scholars, and allied wellbeing and fitness care execs within the neurosciences who require a speedy, readable evaluation of a fancy subject.

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Concise Neurology

Concise Neurology provides neurology content material in a logical series inside each one of ten chapters. almost each subject of curiosity to clinicians is roofed in a single or pages to facilitate effective overview of the middle options in analysis and therapy for every syndrome or sickness. the writer has maintained a thematic cohesion throughout comparable pages, which permits for random analyzing to be virtually as fruitful as sequential studying.

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They are located mainly in the forebrain and are the main source of EPSPs in the CNS (seizurogenic). AMPA receptors agonized by β-N-oxalyl aminoL-alanine (BOAA) can lead to lathyrism (spastic paraparesis caused by chronic chickpeas ingestion). The kainate r eceptors are localized in primary afferent C-fibers in the spinal cord. The indir ectly gated (metabotr opic) mGluR1-8 r eceptors, coupled to second messengers cAMP and PLC, are involved in synaptic plasticity, learning, memory, and neuroprotection.

0 mmol/L. 0 mmol/L (rhabdomyolysis). Digoxin toxicity is increased by hypokalemia. Tetany is seen during alkalosis, which decreases ionized calcium by binding calcium to proteins. Paradoxically, when hypocalcemia is present, hypokalemia protects against tetany. Corr ection of hypokalemia can pr ecipitate hypocalcemic tetany. Magnesium deficiency causes reduced intracellular Kϩ (impaired Naϩ/Kϩ-ATPase) and renal Kϩ wasting. Transcellular Shift β2-sympathomimetics (albuterol, terbutaline, metaproterenol, ephedrine, pseudoephedrine, phenylpropanolamine) xanthines (theophylline, caffeine), verapamil intoxication, chloroquine intoxication, insulin overdose Increased Renal Loss Diuretics (acetazolamide, thiazides, furosemide), mineralocorticoids (fludrocortisone), high-dose penicillin and glucocorticoids Through Mg+ depletion: cisplatin, aminoglycosides, amphotericin B Disorders: hyperthyroidism, familial hypokalemic periodic paralysis, high-epinephrine conditions Metabolic alkalosis due to vomiting, hyperaldosteronism, and Cushing’s distal renal tubular acidosis Kϩ replacement should be given at a rate ≤20 mmol/h in glucose-free solutions with cardiac monitoring.

Myotonia of eyelids and limbs is present between attacks. Progressive vacuolar myopathy develops over time. Respiratory muscles are spared. • Paramyotonia congenita (PMC) is characterized by myotonia during or after exercise or exposure to cold. Interictally, facial, eyelid, and pharyngeal myotonia may be present. EMG shows myotonic activity and decreased CMAP amplitudes with muscle cooling. • Potassium-aggravated myotonia is not affected by cold exposure. Chloride Channels (Autosomal Dominant and Recessive; CLCN1, 7q) • Myotonia congenita (AD: Thomsen disease; AR: Becker myotonia ) results in attacks of myotonia triggered by forceful muscle contraction after a period of rest.

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