By Reinhard Rohkamm

Nordwest-Krankenhaus Sanderbusch, Sande, Germany. Pocket atlas bargains top of the range colour illustrations and distinctive info at the prognosis of signs and symptoms of neurologic ailment. Emphasizes stipulations most often visible in scientific perform. additionally contains the basics of the anatomic constitution of the worried process. Softcover.

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Extra info for Color Atlas of Neurology (Flexibook)

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Radicular syndrome. A lesion of a single ventral nerve root (caused, for example, by a herniated intervertebral disk) produces weakness in the associated myotome. Muscles supplied by multiple nerve roots are only slightly weakened, if at all, but those supplied by a single root may be frankly paralyzed and atrophic (segment-indicating muscles, cf. Table 2, p. 357). Involvement of the dorsal root produces pain and paresthesia in the associated dermatome, which may be triggered by straining (sneezing, coughing), movement (walking), or local percussion.

Posterior spinal a. Spinal arteries Anterior spinal a. and v. Posterior spinal v. Sulcocommissural a. Anterior radicular v. Spinal veins Posterior external vertebral venous plexus Vasocorona Epidural space Pia mater Ventral root Spinal nerve Spinal branch Spinal ganglia Vessels of spinal cord (left: arteries; right: veins) 23 Argo light Argo Anatomical and Functional Organization Anatomical and Functional Organization Cortical Structures 24 Different areas of the cerebral cortex (neocortex) may be distinguished from one another by their histological features and neuroanatomical connections.

Structures: Most of the cerebellar hemispheres, including the declive, folium, and tuber of the vermis. Afferent connections: From sensory and motor cortical areas, premotor cortex, and parietal lobes via pontine nuclei and the inferior olive. Efferent connections: From the dentate nucleus to the red nucleus and the ventrolateral nucleus of the thalamus, and from these structures onward to motor and premotor cortex. Functions: Coordination, speed, and precision of body movement and speech. Effects of lesions: Delayed initiation and termination of movement, mistiming of agonist and antagonist contraction in movement sequences, intention tremor, limb ataxia.

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