By Antonio Mancini

It is a ebook that makes an attempt to suggest itself as a brand new set off within the extensive international of anorexia nervosa. The originality of its suggestion is composed in imminent anorexia nervosa, not just by means of endocrinological and mental views, but additionally by means of anthropological, philosophical and moral standpoint. during this approach it is not purely an replace of particular literature, but in addition integration with a brand new approach to research this situation. the aim of the booklet is to process anorexia nervosa from various issues of view, to arrive a brand new interpretation which contains notions from organic and human sciences interpreted in a special version and which may let a brand new approach to remedy.

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Eur J Endocrinol, 2002, 147, 275-286. Gerald, C; Walker, AW; Criscione, L; Gustaffson, EL; Batzl-Hartman, C; Smith, E; Vaysse, P; Durkin, MM; Laz, TM; Linemeyer, DL; Schaffhauser, AO; Whitebre, AD; Hofbauer, KG; Taber, RI; Branchek, TA, Weinshank, RL. A receptor subtype involved in neuropeptide-Y induced food intake. Nature, 1996, 382, 168-171. Rosenkranz, K; Hinney, A; Ziegler, A; von Prittwitz, S; Barth, N; Roth, H; Mayer, H; Siegfried, W; Lehmkuhl, G; Poutska, F; Schmidt, M, Schafer H, Remschmidt, H, Hebebrand, J.

Leptin levels in patients with anorexia nervosa are reduced in the acute stage and elevated upon short-term weight restoration. Mol Psychiatry, 1997, 2, 330–334. [92] Kadowaki T, Yamauchi T, Kubota N, Hara K, Ueki K, Tobe K. Adiponectin and adiponectin receptors in insulin resistance, diabetes and the metabolic syndrome. J Clin Invest, 2006, 116: 1784-1792. [93] Franssila-Kallunki, A; Rissanen, A; Ekstrand, A; Eriksson, J; Saloranta, C; Widen, E; Schalin-Jantti, C; Goop, L. Fuel metabolism in anorexia nervosa and simple obesity.

These findings may explain the difficulty patients with anorexia nervosa have in gaining weight and may provide the underlying mechanism for the neuroendocrine abnormalities seen in patients with anorexia nervosa or strenuously exercising women. 22 A. Mancini, V. Di Donna, E. Leone, et al. Mutational analysis of the coding region and part of the promoter region of the leptin gene in patients with AN has yielded negative results, suggesting that involvement of this gene in the etiology of AN is unlikely (see review, ref.

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